6 OKTOBER, 2022 / STUREBLOMBERG (original swedish article)
ENGLISH


To keep the body intact for a longer time after death, the body is sometimes embalmed. This involves an embalmer draining the blood from the veins of the deceased and injecting preservative and coloured fluids, such as formalin, in its place, making the deceased look more alive in the coffin at the funeral. The liquid is injected through one of the deceased's body veins, usually in the groin, which slows down the decomposition process.

In Sweden, this method is not as common as in countries such as England and the USA. In Sweden, the deceased is usually kept in a refrigerated compartment until the funeral. However, embalming also occurs in Sweden, but is less widespread than in other countries.

The embalming procedure itself, involving the draining of blood and the injection of coloured preservative fluids, is usually a simple procedure. However, with the start of Covid-19 vaccination, embalmers began to experience difficulties in injecting the embalming fluids into the vascular system. They then entered the blood vessels with various instruments to possibly fish out anything that was blocking the flow in the blood vessel.

They found hard, white rubber-like strands, sometimes up to 1.20 metres long [3], which could be so thick that they almost completely blocked the flow in the blood vessel. Such long hard strands are now found in 80% of deceased people vaccinated against Covid-19 but not in unvaccinated people, according to embalmers Richard Hirschman [1] and John Olooney [2].

In their long 20-year careers as embalmers, they have never seen anything like this, suggesting that this is a result of the vaccinations. The findings are very worrying considering how many people have actually been vaccinated against Covid-19. However, it is not certain that all vaccinated people are affected. Many embalmers have contacted Richard Hirschman [1] and John Olooney [2] about their similar experiences, but do not want to put their names on the record because of the risk of being hanged and losing their jobs. Below are pictures of what these white, hard strings look like:

1. This white, hard and rubbery string of probably "denatured" proteins was fished out by the American embalmer Richard Hirschman from a deceased man from the large blood vessel in the neck that carries blood from the heart to the brain, the arteria carotis interna. Judging by its thickness, it may have completely blocked the flow of blood to the brain.

 

2. These 7 test tubes contain extensive white, hard, rubber-like strands from 7 different deceased persons. If you enlarge the image, you can see that tubes 3 and 4 in particular contain an enormous amount of such material, which may have blocked the flow of blood throughout the body

 

3. Here, English embalmer John Clooney holds up a whole glass of the hard, white, rubbery protein strands from a single patient. The top one was fished out of the aorta, which is the main artery of the body.

 

4. The test tube held up by pathologist Dr Ryan Cole from Idaho, USA, contains exactly the same type of white, hard rubber-like strands that fish out of the blood vessels we autopsied from a vaccinated patient.

 

5. This image shows that these strands can be long and connected to several strands from other blood vessels. The image also shows ordinary blood clots on the surface of the white, hard, rubbery strands.


Cause not fully understood

The cause of the formation of these white, hard, rubber-like plugs is not yet fully understood. Several researchers believe that they are amyloid, which are protein molecules in the blood plasma, such as fibrinogen, that continually stick to each other until eventually the whole blood vessel becomes blocked and the blood supply to various organs stops.

This can lead to organ failure and death. It is unclear how long people can live with these long rubber-like clots in their bloodstream. Pathologist Dr Ryan Cole from Idaho in the US [3] painted a picture of a long string attached to the wall of the blood vessel at one end, where it is formed and blood to the various organs flows around and past this string. He argued that people will live as long as the blood supply to the various organs is sufficient.

As the clot or cord builds up over time, blood circulation becomes increasingly poor until the blood supply becomes insufficient, at which point death can occur [3]. This also means that if children are affected, the time to total vessel blockage and death is shorter because children have much narrower blood vessels.

 

The ordinary coagulation process

The fact that bleeding associated with wound injuries stops after a certain time is well known to all people. Medical science has mapped out exactly how this happens and what factors are involved. The platelets are essential for the whole haemostasis procedure to get under way. If you do not have enough platelets, the bleeding will not stop.

Once the platelets have started the haemostasis process, the coagulation process begins, which involves the formation of a network of fibrin (cf. fibres) that traps the red blood cells and prevents them from leaking out. The coagulation process is very complex and at least 13 different factors must interact to form this fibrous network, known as the "coagulation cascade". When the wound is healed, these fibrin threads are cut by "plasmin", which is called "fibrinolysis" ("lys" ung. = "dissolve"). What remains in the blood are small pieces of fibrin, called D-dimer.

If a laboratory test shows elevated levels of D-dimer, this means that there are blood clots in the circulatory system. The most worrying thing is if these blood clots are transported from, for example, the calves to the lungs and get stuck there, so-called pulmonary embolism, which can be associated with immediate death. In the case of elevated D-dimers, lung X-rays with MRI camera and ultrasound of blood vessels in the calves are therefore routinely performed. Several emergency physicians [5] have found sky-high D-dimer values in almost all patients vaccinated against Covid-19, but no large blood clots in the calves or lungs were seen [4].

It has been concluded that the clots were small, but scattered throughout the system, so-called disseminated intravascular coagulation (DIC). But in addition to this widespread formation of common blood clots in people vaccinated against Covid-19, these white hard, long rubber-like clots, as shown above, are also formed, which is very worrying.

 

Amyloid - a type of coagulation that is normally rare

When proteins bend or 'fold' incorrectly for various reasons, called 'denaturation', they lose their natural function. They can also clump together, forming amyloid, which can cause various diseases. Amyloid fibrils (a smaller form of a fibre) are insoluble nanofibres of protein, which form and accumulate spontaneously, producing amyloid plaques.

It may consist of a single type of protein or of several, with the individual protein molecules lying perpendicular to the direction of the fibril, and held together by hydrogen bonds. They are thus built up in the longitudinal direction. If several fibrils clump together, the result is long strands like those shown in the pictures above. The amyloid is self-generating and just grows and grows while being enormously difficult to break down.

 

Amyloid can be caused by various pathogens

Different pathogens can cause proteins to denature and clump together. 1. the malaria parasite (Plasmodium falciparum) enters the red blood cell via the CD147 receptor [6, 7]. the parasite feeds on the amino acids in the haemoglobin of the red blood cells [8] and multiplies in this way.

When the blood cells burst, the new parasites spread to other blood cells and the whole body becomes infected with malaria parasites. However, with certain mutations of the haemoglobin molecule, the haemoglobin molecules clump together, forming so-called Heinz bodies, which are denatured haemoglobin in the red blood cells [9].

It has been shown that there are also amyloidogenic peptides (=amino acid sequences) encoded in Plasmodium falciparum, which play a role in the pathology of malaria [10].

2. Bacteria such as Leishmania, Entamoeba and Tryptanosoma can cause the blood cell to rupture primarily and the bacteria can then absorb all the amino acids of haemoglobin as well as iron and haem from the blood cell [11].

3. It is also known that SARS-CoV-2, for example, enters the red blood cell via the same receptor, CD147 [12, 13] and it has been suggested that viruses can also cause haemoglobin to dissociate and clump together [14].

 

Amyloid can be caused by prions

Prions are proteins that contain a special "prion" sequence of amino acids and therefore become misfolded, causing clumping of different proteins. Prions became known as the cause of "mad cow disease" but are also associated with other neurodegenerative diseases such as Alzheimer's disease. Prions also prevent other proteins from folding or unfolding correctly, which gives rise to an auto-catalytic ability and means that more and more proteins will unfold incorrectly, resulting in denaturation and clumping of proteins. The prions also cannot be broken down, so there is a progressive deterioration over time.

 

Extensive amyloid formation can be caused by spike proteins

Several years ago, the South African researcher Etheresia Pretorius demonstrated that fibrinogen in blood plasma could clump together to form amyloid, which was resistant to fibrinolysis [15, 16]. She then found the same amyloid-like fibrin strands in platelet-poor plasma from patients with post-Covid (a). When Pretorius et al. added spike protein (its S1 part) to platelet-free plasma from healthy subjects in a later experiment (b), they obtained the same result: amyloid that was resistant to fibrinolysis.

It is known that the SARS-CoV-2 spike protein has prion-like peptides, which may explain the misfolding of the haemoglobin and the formation of amyloid. These experiments show that it is possible to form clots in the bloodstream that are resistant to degradation and that are completely independent of natural blood coagulation and that can be triggered by spike protein alone. But the spike protein does not only attack the fibrinogen in the blood plasma.

SARS-CoV-2 has also been shown to attack the haemoglobin inside the blood body and Heinz' bodies - as in malaria - have been found in 14 patients with Covid-19 [17, 18]. In addition, during the breakdown of the blood cell (hemolysis), amyloid fibrils are also formed in plasma from hemoglobin [19]. This could explain the size and enormous spread of the amyloid clots.

Blood plasma contains only c;a 1.5 g/L of fibrinogen but large amounts of haemoglobin are present in the red blood cells (≈ 2.2 L), which consist practically only of haemoglobin for oxygen transport. Covid-19 vaccinations have been shown to be associated with large-scale red blood cell decomposition (haemolysis) [20] whereby globulins, iron and and haem, which normally bind the iron, are released into the blood plasma. In addition to these white, hard, rubbery strands described above, this can cause pathological deposition of iron in various organs ("haemochromatosis"), as well as the disease "porphyria".

Porphyria is a so-called "northern" hereditary disease, now found in 200,000 Americans, caused by a mutation in an enzyme needed to form the heme needed for haemoglobin to carry oxygen. This disease can intermittently cause severe abdominal pain. To give an idea of the extent of haemolysis, since the start of the Covid-19 vaccination, 172 times more cases of profyri have been reported than 5 years previously. And 74% of these are reported in the first 24 hours after injection, suggesting that vaccination is the cause of red blood cell breakdown - and probably also its sequelae.


 

References

see previous page No 159, at the end of the swedish version !


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